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Antibodies against PTM of beta-cell autoantigens, such as GAD65 and insulin, may play a critical role in disease onset and development

Moderate instability promotes adaptation by enabling oncogene amplifications (e.g., ERBB2, CCND1, MYC) or loss of tumor suppressor loci, facilitating therapeutic escape, whereas excessive instability compromises viability

Documented observations within controlled laboratory research models Recorded observations in experimental models are primarily associated with glutathione-related redox signaling, oxidative stress markers, and intracellular antioxidant response behavior

Inducible nitric oxide synthase during the late phase of sepsis is associated with hypothermia and immune cell migration