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Furthermore, PCBs have been shown to accelerate atherosclerosis through activation of the aryl hydrocarbon receptor (AHR), upregulation of adhesion molecules, production of ROS, endothelial dysfunction, and apoptosis in vascular cells [53]

Ultimately, terminal membrane rupture releases DAMPs (High Mobility Group Box 1, HMGB1, Lactate Dehydrogenase, LDH) and cytokines (Tumor Necrosis Factor, TNF, IFN-), while permanently impairing cross-presentation capability