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Description

Increased delivery of NEFA to the liver leads to hepatic steatosis, and facilitate alcohol-induced liver injury by activating KCs [215]

Metabolism: The liver identifies the compound and starts breaking it down

Intriguingly, we revealed that FSP1 inhibition alone was sufficient to trigger ferroptosis in a subset of hepatocellular carcinoma (HCC) and cholangiocarcinoma (CCA) cells