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In conclusion, ferroptosis plays a crucial role in the pathogenesis of NAFLD/NASH and offers novel therapeutic and predictive avenues for patients

Direct involvement of the P2X7 purinergic receptor in increased oxidative stress through stimulation of this receptor by extracellular -synuclein was shown on microglial cells (Jiang et al., 2015)

Santos BL, Oliveira MN, Coelho PL et al (2015) Flavonoids suppress human glioblastoma cell growth by inhibiting cell metabolism, migration, and by regulating extracellular matrix proteins and metalloproteinases expression