slc25a39 is necessary for mitochondrial glutathione import in mammalian cells

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated transport Dual regulation of SLC25A39 by

Name: slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated transport Dual regulation of SLC25A39 by
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Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis ScienceDirect glutathione and mitochondrial function SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells Autoregulatory control of mitochondrial glutathione homeostasis Science Glutathione driven redox decisions in cell survival and death

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Afterwards, the controls, such as PC not spiked with GX-G3 or unrelated comparators, were analyzed in comparison with the spiked samples

These treatments decreased cell viability and increased both soluble and lipid reactive oxygen species (ROS) generation but did not affect mitochondrial ROS or mitochondrial mass