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doi: 10.1016/j.ijsu.2022.106936

Future platforms may integrate anti-NGF agents, endothelial-targeted aptamers, nerve fiber blocking peptides, and exosome carriers to build next-generation interventions that address the vascularnervepain axis and jointly optimize structural repair and analgesia

DEFINITION AND CLINICAL PHENOTYPE OF DbCM Numerous mechanisms have been identified and proposed to contribute to the pathogenesis of DbCM, including oxidative stress, fibrosis, inflammation, various forms of cell death, alterations in myocardial energetics, among others [2]