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(2024b) demonstrated that through the inhibition of inflammatory macrophages, regulation of extracellular redox homeostasis, and downregulation of the MAPK/ERK signaling pathway, it is possible to suppress over-activated inflammation and restore cell-tight junction proteins

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This clinical evidence collectively supports the notion that the maintenance of uterine fibroids may depend on a stably established E-P-OS network within their local microenvironment, which retains the capacity for self-activation and reconstruction even after short-term hormonal suppression

By comparison, GOx/CuSAE exhibited greater activity of NADPH depletion, indicating the synergistic effect of glucose decrease in NADPH depletion of the hybrid nanozyme