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Intriguingly, under conditions of caspase-1 deficiency or pharmacological inhibition, an alternative pathway emerges: the inflammasome redirects its signaling to activate caspase-3, which subsequently processes GSDME to execute pyroptosis [80]

Arantes LAM, Aguiar CJ, Amaya MJ et al (2012) Nuclear inositol 1,4,5-trisphosphate is a necessary and conserved signal for the induction of both pathological and physiological cardiomyocyte hypertrophy