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New antifibrotic treatments indicated for idiopathic pulmonary fibrosis offer hopes and raises questions

STAT3 and STAT5 have been reported to act by inhibiting the antitumor immune response by activating, at least in part, the production of inflammatory cytokines (IL-1, IL-17, IL-10, TGF-, or VEGF) and promoting tumor growth and metastasis (150)

Furthermore, PKA phosphorylates and regulates the activity and expression of the cAMP-response element modulator (CREM) protein family, a group of transcription factors playing an important role in various cellular functions (82)

While curtailing lactate mitigates lipid peroxidation, sparing membranes from 4-HNE assault, it risks starving cells of ATP in regions where mitochondria are irreparably damaged [210]