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Therefore, blocking the activity of Syt2 should prevent sudden massive mucin release without impairing slow, steady baseline mucin secretion that is required for airway health. In this study, a collaborative effort between MD Anderson, Stanford Medicine and Ulm University, the researchers verified Syt2 as a viable therapeutic target protein in several types of preclinical models

The result of mapping served to determine the gene expression levels based on the TPM (Trancripts per Million)

Adherence to local and international safety protocols, including SDS handling and PPE

The complexity of nanoparticle dissolution and its importance in nanotoxicological studies