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Although both PKC and PKA have been reported to be the upstream kinase for Ser184 of GSTP in MGR3 human glioblastoma cells (51), our study indicated that only cPKC interacted with GSTP and mediated LPS-stimulated GSTP phosphorylation

These results suggested that LAbs@Lip@BMn/Rapa treatment effectively alleviated neuronal damage and provided excellent neuroprotection in the ATau copathogenic AD mouse model