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Unlike other forms of regulated cell death, ferroptosis is an iron-dependent, non-apoptotic process [26, 27] characterised by increased reactive oxygen species (ROS) and lipid peroxidation, both of which are driven by the accumulation of intracellular iron [28,29,30]

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Thus, the integrity of RBCs and protein homeostasis regulate iron-catalyzed H 2 S, most likely through sequestration of iron into the bound vs