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and exposure to ticks or areas where Lyme disease is endemic

While they do not cause cancer, their mechanism could theoretically support the blood supply to a tumor

Integrating toxicokinetic data, mechanistic evidence, and exposure science therefore provides a more reliable framework for evaluating risks and distinguishing genuine causal relationships from statistical correlations arising from bias, co-exposures, or measurement limitations

Molecular mechanisms regulating NPPCs in IDD Under conditions of elevated oxidative stress, excessive reactive oxygen species (ROS) accumulation and mitochondrial damage activate senescence pathways like P53/P21, contributing to accelerated ECM catabolism and NPPC senescence [76]