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Fertil Steril (2014) 102(6):157683.e4

While this effect could potentially be explained by the propensity for EGFR to co-amplify with CHCHD2, the amplification of EGFR was not solely associated with a decreased patient survival (Fig

Differential anti-inflammatory and anti-oxidative effects of dexamethasone and N-acetylcysteine in endotoxin-induced lung inflammation

We suggest that mast cells participate in the pathogenesis of AD, and this process could be accelerated and worsened in brain injury, stress, and PTSD comorbidity