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Intradermal & Subcutaneous Routes: Target fibrotic skin and vascular damage seen in scleroderma-like MCTD features

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Secretion of IFN, IL-17, and IL-22 by Th1 and Th17 cells activates B cells, which secrete anti-tTG, anti-neo-tTG, and anti-endomysial autoantibodies (EMA)

To understand how Cellular Therapy and Stem Cells for MCTD can provide relief and regeneration, it is vital to examine the key cellular players involved in MCTD pathogenesis: T Lymphocytes (CD4 and CD8): These immune cells drive inflammation and tissue damage by recognizing autoantigens and releasing destructive cytokines