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Endoplasmic reticulum (ER) stress and hypoxia response pathways interact to potentiate hypoxia-inducible factor 1 (HIF-1) transcriptional activity on targets like vascular endothelial growth factor (VEGF)

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Namely, similar vascular and multiorgan failure presentations (and counteractions with BPC 157 therapy) have been seen with other similar occlusion syndromes and occlusion-like syndromes, after occlusion of the major vessel(s), arteries and veins, both peripherally and centrally, and with similar noxious procedures