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mTOR, a negative regulator of autophagy, integrates signals from nutrient and growth factor pathways to control cellular growth and metabolism and inhibits autophagy induction by blocking the initial inducer of autophagy [211, 212]

These damaged proteins can become joined together through a mechanism called crosslinking, which causes increased stiffness and loss of elasticity in several tissues throughout the body (Nawale 2006

doi: 10.1016/j.immuni.2022.12.003 71 MaJZhangHChenYLiuXTianJShenW

32,33,34,35 For example, it has been shown that histone acetylation is closely associated with gene transcription and the alterations of histone acetylation are tightly associated with cancer phenotypes in multiple cancers