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The study found that FOS/GOS supplementation inhibited the dysregulated expression of intestinal epithelial gap junction proteinsincluding Claudin-1, Claudin-15, Zonula occludens-1 (ZO-1), and Junctional adhesion molecule-Athereby mitigating barrier damage and reversing HFD-induced hyperpermeability

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Such imbalances have been associated with increased susceptibility to infections as well as a higher risk of autoimmune diseases and certain cancers [44]