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Pharmacological activation of aldehyde dehydrogenase 2 protects against heatstroke-induced acute lung injury by modulating oxidative stress and endothelial dysfunction

Alternative mechanisms currently under investigation appear to include potentially peptide-related modulation of energy expenditure and fatty acid oxidation pathways

For example, in the case of ECs, each cell type, either cardiac or vascular, might have a preferential matrix for optimal culturing, and depending on the objective of the study, the microfluidic composition and dynamics must also be adjusted [270]

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