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It has been shown that pathogenic bacteria or viruses in inflamed tissues promote ferroptosis in cells while activating immune cells and triggering inflammatory responses through the action of lipopolysaccharides, peptidoglycans, bacterial or viral DNA, and host cell toll-like receptors carried or released by themselves (60)

(2017) also reported the accumulation of some GST proteins in systemically infected leaves of soybeans susceptible to mungbean yellow mosaic India virus (MYMIV) and mungbean yellow mosaic virus (MYMV)

To examine the incorporation of GSH-derived cysteine into downstream pathways, we used an inverse stable-isotope labelling approach 34,35

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