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As with BPC-157, the bulk of supportive evidence comes from animal models, knee-specific human data is sparse, and it is not approved for therapeutic use in Australia

Smith R M, Martell A E

The progressive decline in mitochondrial function, characterized by impaired ATP synthesis, excessive ROS production, and structural abnormalities, triggers a vicious cycle involving energy crises, oxidative stress, and inflammatory responses, ultimately accelerating cardiomyocyte death [129, 130]

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