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Insulin is the dominant metabolic hormone controlling glucose and lipid homeostasis, such that humans or mice with insulin deficiency or mutations of the IR develop severe hyperglycemia and uncontrolled lipolysis (810), whereas animals lacking IGF-1 or with mutations in IGF1R display severe growth deficiency both prenatally (11) and postnatally (12, 13)

The obesogen hypothesis proposes that prenatal exposure reprograms stem cells toward preferential adipocyte differentiation, potentially establishing transgenerational inheritance patterns (222)

One must raise awareness, promote safe treatments, and protect patients from risks

Ellul-Micallef R