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544 In addition, emerging targets such as IL-36 pathway inhibitors promote Treg cell proliferation by neutralizing the proinflammatory effects of IL-36, and TLR4/MyD88 small molecule inhibitors provide a new way of treating UC by modulating innate immune hyperactivation triggered by colony dysregulation

Overexpression of the nuclear factor (erythroid-derived)-like 2 (NFE2L2) or suppression of the Kelch-like ECH-associated protein 1 (Keap1) has been shown to promote glioblastoma invasiveness and hence accelerate glioma growth (72)

Specifically, part of the participants received a second intervention 1 year after the initial treatment of 3 consecutive months

doi: 10.1016/j.jhazmat.2016.03.015