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The cellular anti-inflammatory action of the nanosheets is achieved by scavenging intracellular ROS of M1 macrophages and promoting their transition to M2 phenotype, which largely prevents mBMSC apoptosis, while the Zn 2+ released from the degrading nanosheets upregulates the ALP level of mBMSCs, facilitating osteogenic differentiation and biomineralization

Dobberfuhl, T

Liao S, Zhou J, Wang H, Chen X, Wang H, Wang G (2013) Arsenite oxidation using biogenic manganese oxides produced by a deep-sea manganese-oxidizing bacterium, Marinobacter sp

Additional research context on copper peptides can be found in our guide to GHK-Cu (Copper Peptide) Now Available: What Researchers Need to Know About This Tissue Repair and Anti-Aging Compound