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Since IGF-1R inhibition results in suppression of downstream AKT activation, the IGF-1R inhibitors (BMS-754807 and GSK1838705A) efficiently inhibit AKT phosphorylation, indicating that the drugs function properly

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Consequently, the C 60 and GHB interactions are weak physisorption, which is also supported by the NCI/RDG analysis showing mostly diffuse dispersion forces (Table 7)