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Excessive iron loading increases the expression of M1-associated inflammatory mediators, including IL-6, TNF-, and IL-1, while suppressing M2 markers such as transglutaminase 2 (TGM2), so efficiently guiding polarization toward the pro-inflammatory M1 phenotype (97)

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Instead of just addressing one aspect of healing, this stack coordinates a multi-faceted approach, from laying the foundational groundwork for new tissue to managing the cleanup crew

Spinal glia division contributes to conditioning lesion-induced axon regeneration into the injured spinal cord: Potential role of cyclic AMP-induced tissue inhibitor of metalloproteinase-1