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Although there are promising novel therapeutic approaches, further research is needed to better understand the disease mechanisms, develop therapies that target difficult to reach tissues and create adjuvant therapies that address secondary cellular processes caused by the enzyme deficiency

Acute kidney injury due to rhabdomyolysis and renal replacement therapy: a critical review

As expected, overexpression of WT GCLC or its desuccinylation-mimicking mutants (K38R, K126R, K326R or 3KR) in U2OS cells significantly enhanced the cellular GSH levels compared to empty vector control (NC) (Fig