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In the CNS, neuroinflammation is propagated through infected monocytes, which promote the release of pro-inflammatory cytokines, such as IL-6, IL-8, IL-10, IL-2R, and TNF-, and stimulation of microglia, contributing to neuropsychiatric symptoms resulting from COVID-19 (Fig

Through the use of a genetically encoded and mitochondrially targeted biosensor 35 , we determined that the mitochondrial GSH pool was more reduced under cystine deprivation relative to replete conditions (Fig

Initial attempts with mammalian liver microsome preparations and recombinant enzymes did not result in appreciable yield