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According to the A-tau interaction hypothesis, A and tau pathology, which results in synaptic dysfunction, neuronal death, and cognitive impairment (96), is determined by the intrinsic interaction with A and tau (A42 enhances tau phosphorylation, truncation, and aggregation, while tau can increase A synthesis)

J Neurosci Res 83(3):476488 Sio YY et al (2023) The ERBB2 exonic variant Pro1170Ala modulates mitogen-activated protein kinase signaling cascades and associates with allergic asthma