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Oligodendrocyte death and Myelin loss in the Cuprizone model: an updated overview of the intrinsic and extrinsic causes of Cuprizone demyelination

Abstract The underlying pathophysiology of Parkinson's disease is complex, but mitochondrial dysfunction has an established and prominent role

The results of GSH therapy could include down regulation of 12- and 15- lipoxygenases, up-regulation of GSH peroxidases, and down regulation of the NFKB cascades that include the induction of Cox-2

TGF- inhibitors, such as pirfenidone or losartan, which are already in clinical use for conditions like IPF, 500 could be repurposed to manage PH by mitigating EndMT and the inflammatory processes contributing to vascular remodeling