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Abstract The anti-oxidative enzyme, glutathione peroxidase 4 (GPX4), helps to promote inflammation resolution by eliminating oxidative species produced by the arachidonic acid (AA) metabolic network

Advanced pharmacological elucidation illuminates the metabolic elegance of ketoacid analogues, whichthrough their sophisticated transamination chemistrysequester excess amino nitrogen reservoirs, channeling them toward de novo essential amino acid biosynthesis

1 Introduction Autoimmune diseases are a heterogeneous group of disorders caused by the misdirection of the immune system toward its host, and the presence of autoantibodies is a common feature of autoimmune diseases ( Given the limited understanding of the complex molecular mechanisms underlying autoimmune diseases, it is widely accepted that a combination of genetic predisposition and environmental factors disrupts immune tolerance (6), This disruption leads to the clonal expansion of autoreactive B and T cells, resulting in the production of autoantibodies that trigger autoimmune inflammation and the pathological attack on the bodys tissues

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