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ROS-induced mtDNA damage impairs mitochondrial OXPHOS, forcing tumor cells to rely on glycolysis for ATP production ( Figure 1 2.1 Mitochondrial metabolic regulation of immune cells T cells rely on OXPHOS and fatty acid oxidation in the resting state, but switch to aerobic glycolysis and fatty acid synthesis upon activation to support proliferation ( Figure 2 )

It should be noted that we do not include mitochondrial glutathione, which the Jaeschke group has found to be fundamental in APAP pathophysiology [36]

B3 (Niacin) Niacin contributes to normal energy release and supports the health of the nervous system