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Patients were excluded in case of acute myocardial, mesenterial or digital ischemia before the initiation of AVP

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Knockdown of NNMT in adipocytes was associated with increased NAD+ levels and altered expression of genes involved in energy expenditure, providing the rationale for pharmacological NNMT inhibition [2]

This approach makes intuitive sense because the amount of incretin stimulation needed to maintain a lower weight is probably less than the amount needed to achieve that lower weight in the first place