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Immune checkpoint molecules, tumor-associated macrophages, regulatory T cells, and myeloid-derived suppressor cells can attenuate the efficacy of ferroptosis and cuproptosis inducers, thereby forming a drug-tolerant microenvironment that promotes therapeutic resistance and early recurrence

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Pro-oxidative activities and dose-response relationship of ()-epigallocatechin-3-gallate in the inhibition of lung cancer cell growth: A comparative study in vivo and in vitro

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