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Conclusions Our data indicates that GSH participates in retarding apoptosis in antihormone-resistant human breast cancer cells and that depletion of this molecule by BSO may be critical in predisposing resistant cells to E 2 -induced apoptotic cell death

Furthermore, the adenosine receptor A2aR, which is expressed on the surface of activated T cells, can be activated by adenosine and has been shown to dampen proximal T cell receptor signaling (102), diminish IL2-dependent proliferation of T cells (103), attenuate T cell cytotoxicity and cytokine production, induce T cell anergy and expression of inhibitory receptors (104), and promote the generation of regulatory T cells (105)

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Another example in this category is methylisoeugenol which had a NIST score of 660