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1, its antitumor efficacy is driven by pleiotropic actions at both molecular and cellular levels: downregulating pro-survival factors (e.g., Bcl-2, Bcl-xL), inhibiting proliferation pathways (e.g., NF-B, PI3K/Akt/mTOR, STAT3), inducing cell cycle arrest, and promoting programmed cell death [35, 36]

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2.3.6 A1AT and SARS- CoV-2 As observed in the case of the Middle East Respiratory Syndrome (MERS) caused by the coronavirus (122), A1AT has been suggested to that A1AT plays a role in protecting against SARS-CoV-2 infection

Regulation of cell proliferation by interleukin-3-induced nuclear translocation of pyruvate kinase