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Hypochlorous acid generated by myeloperoxidase modifies adjacent tryptophan and glycine residues in the catalytic domain of matrix metalloproteinase-7 (matrilysin): An oxidative mechanism for restraining proteolytic activity during inflammation

[13] Most of these reactions, if they occur, subside as the body adjusts to therapy

Electroconvulsive therapy mediates neuroplasticity of white matter microstructure in major depression

Recent studies have demonstrated that BMAL1s role extends beyond regulating and oscillating core clock genes to include maintaining redox balance and enhancing cell viability under oxidative conditions