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Reducing small intestinal permeability attenuates colitis in the IL10 gene-deficient mouse

Inflammatory modulation: animal models of experimental colitis (TNBS, DSS), toxic hepatitis and joint damage documented reduction of pro-inflammatory cytokines (TNF-, IL-6, IL-1) and improvement of histological lesion scores

Diabetic neuropathy is a common complication of T2DM, and GSTP1 gene polymorphism may contribute to the development of T2DM (140,141)

While the precise mechanisms linking these metabolic alterations to fibrotic remodeling remain to be fully elucidated, current studies strongly support amino acid metabolism as a central regulatory axis in fibrosis initiation and progression