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Research findings indicate that Donafenib can downregulate the expression of ferroptosis resistance factors SLC7A11 and GPX4, enhancing ferroptosis and apoptosis in hepatic cells, thereby demonstrating its therapeutic potential [256]

In contrast, in vivo gene therapy involves the direct delivery of therapeutic agents to the patients body via systemic or local routes

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The impairment of MAGMAS function in human is responsible for a severe skeletal dysplasia