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The shift towards H 2 S-producing pathways may be driven by down-regulation of the aerobic oxidative degradation of cysteine to taurine, mediated by cysteine dioxygenase, as evidenced by the lower taurine/cysteine ratio in arterial cord blood, not only in severe IPTB cases, irrespective of treatment, but in NAC-treated moderate IPTB cases as well

Additionally, understanding genetic variants in glutathione metabolism (such as GSTM1 and GSTP1 status) can support a more informed conversation with your provider about whether combined antioxidant and GLP-1 therapy aligns with your metabolic profile

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