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Treatment and management of neuroinflammation Low-dose naltrexone (LDN) As a potent antagonist of central -opioid receptors (OR), naltrexone at low doses (LDN, 35 mg) acts through the blockade of these receptors to consequently reverse their inhibition of transient receptor potential cation channel subfamily M member 3 (TRPM3)

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Other contributors include faulty axonal transport, resulting in buildup of neurofilaments and synaptic breakdown (Taylor et al., 2016), and genetic mutationssuch as SOD1 (causing harmful protein accumulation), FUS (interfering with RNA handling), and TBK1 or OPTN (which impair the cell's waste disposal system known as autophagy