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PMID: 39298550 Exp Cell Res Molecular mechanism of MAFB transcriptional activation of PPARD in regulating adipose browning and protecting against vascular endothelial cell injury

Under lipotoxicity, FFA inhibition of lysosomal function blocks autophagic flux, which causes the accumulation of damaged mitochondria as well as of lipids digested by lipophagy, thereby exacerbating the decline in mitochondria and cellular functions, which leads to progressive disease 8,10,11,12,13,14,15,16,17,18

This may also be a consequence of underlying assumptions of medium homogeneity underlying conventional attenuation-based analysis, even after accounting for overlying tissue, and high sensitivity of attenuation-based outcomes to shadowing 30, 67

Expression, characterization and tissue distribution of a new cellular selenium-dependent glutathione peroxidase, GSHPxGI