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Collectively, our study demonstrates that SLC1A4 mutations lead to dysregulation of multiple solute carrier protein genes causing metabolic stress, excitability defects, and synaptic abnormalities, providing a molecular framework for understanding SLC1A4 -related neurodevelopmental disorders and identifying potential therapeutic targets

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Future studies will measure PFOS partitioning in other organs of interest, such as the liver and kidney, to further understand lifestyle factor influences on PFOS distribution