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A particular point mutant of the protein, A53T, associated with familial PD is also known to fibrillate at a significantly higher rate than the normal protein (Coskuner and Wise-Scira, 2013) owing to its increased propensity to form beta fibrils as well as by partially negating the effect of long-range interactions between the N and C terminus

Numerous structurally diverse foreign substances and endogenous compounds can induce several CYP genes, resulting in increased synthesis and activity of the corresponding enzymes and acceleration of the drug-substrates metabolism

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This damage can accelerate the aging process and increase the risk of chronic diseases such as cardiovascular issues, diabetes, and even neurodegenerative conditions like Alzheimers