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doi: 10.1523/JNEUROSCI.18-21-08900.1998 Pubmed Abstract CrossRef Google Scholar View reference in article 20 GeinismanY.de Toledo-MorrellL.MorrellF

[8] This supports the involvement of ERK1/2 under the study conditions, but it does not prove that ERK1/2 is the only pathway involved or that it sits directly below FAK in a single BPC-157 signaling chainthe FAK and ERK findings came from different experimental systems

Researchers study whether certain compounds can maintain telomere length by activating the enzyme telomerase

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