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We identified a significant epistatic interaction between GSS rs3761144 and GSTA1 rs3957356 on urine-Hg levels ( P = 0.015, P perm = 0.017)

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Additionally, pathway enrichment analysis further revealed that these alterations corresponded to disruptions in key metabolic routes, including the glucose-alanine cycle, glucose metabolism, beta-oxidation of very long-chain fatty acids, glutathione metabolism, transfer of acetyl groups into mitochondria, and the degradation pathways of branched-chain aminoacids (BCAA) (Figure S3)