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Thus, on one side, there may be a potential dosing window to kill malignant hematopoietic cells by targeting GPX4, leaving healthy HSPCs intact

The inhibition of APE1 expression or activity impacted the cell cycle, cell proliferation, colony formation, and apoptosis of cancer cells [30, 31]

NRF2 enhances the cells capacity to manage oxidative stress and ionizing radiation, as well as other exogenous and endogenous challenges, by regulating the expression of various antioxidant genes, such as glutathione S-transferase, glutathione reductase, and NAD(P)H quinone oxidoreductase [1]

HIV-1 Tat neurotoxicity is prevented by matrix metalloproteinase inhibitors