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We show that zonulin is a central factor in this process as it disrupts intestinal TJ proteins, enhances intestinal permeability and leads to Th1 and Th17 infiltration in the lamina propria before the onset of murine and human arthritis

Beyond taxonomic shifts, these microbial changes lead to functional alterations in microbial metabolic activity, resulting in modified production of bioactive metabolites that can influence multiple host physiological systems, including the gutovary axis (73, 74)

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