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The enhanced autophagy in drug-resistant cells is mainly due to the destruction of mitochondrial function caused by tamoxifen, and drug-resistant cells meet their energy demand through autophagy

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Petroff D, Blank V, Newsome PN, ShalimarVoican CS, Thiele M, deLedinghen V, Baumeler S, Chan WK, Perlemuter G, Cardoso AC, Aggarwal S, Sasso M, Eddowes PJ, Allison M, Tsochatzis E, Anstee QM, Sheridan D, Cobbold JF, Naveau S, Lupsor-Platon M, Mueller S, Krag A, Irles-Depe M, Semela D, Wong G, Wong VW, Villela-Nogueira CA, Garg H, Chazouilleres O, Wiegand J, Karlas T

Currently, activating key innate immune pathwaysparticularly the cyclic GMP-AMP synthase-stimulator of interferon genes (cGAS-STING) pathwayhas emerged as a promising strategy to enhance antitumor immune responses [8,9,10,11,12,13,14,15,16,17]