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49 Gram-positive bacteria, such as S

Additionally, the role of non-classical ferroptosis regulators (e.g., FSP1, DHODH, and GCH1/BH4) in LN is entirely unexploredwhether these radical-trapping antioxidant pathways compensate for GPX4 deficiency in immune cells or renal tissue, or if their dysfunction exacerbates ferroptosis susceptibility, has not been investigated